Tag Archive | "Armando Vecchietti"

Temperature Variation in Patients Regularly Using A Reich Orgone Accumulator


Temperature readings before and after daily sessions inside an Orgone Energy Accumulator (ORAC) were independently taken by four volunteers. The data was provided to the author and then analysed. Duration of the ORAC use ranged from 8 months to nearly 10 years. Two parameters, the charging capacity and the charging potency were outlined to evaluate any possible effects of the ORAC on the volunteers’ health conditions. Overall, the four volunteers showed improved health conditions and vitality after the experimental period. In one case microscopic and cytologic tests were carried out before starting the daily use of the ORAC and after 8 months of use. Comparison of the results showed a good performance of the ORAC on the red blood cells that changed from an original T-reaction (Reich’s term for lesser vitality indicators) to a B-reaction (his term indicating greater vitality). The Ca I cells (Cancer Type 1) were no longer found on the second exam. Ca II and Ca III cells were still found but without the development of new similar cells.


Over the last period of some decades many efforts were devoted to measurements of the temperature inside and outside an orgone energy accumulator (ORAC), aiming at examining Reich’s original findings on the thermal difference (T0-T) by various researchers [1]. However, many efforts were also extended to evaluate the physiological response of people exposed to the orgone energy fields inside an ORAC. One of the most investigated parameters was the body core temperature. In general, an increase of body core temperature was observed after exposure to elevated orgonomic potentials such as that characteristic within an ORAC. Reich observed an increase as high as 0.8-1.0 °C in patients sitting inside an ORAC for a period:

“…. In 1942 an important phenomenon was discovered which has a decisive connection with the body lumination in the accumulator: Body temperature rises in the accumulator as much as one degree centigrade (the rapidity and amount of increase varying from individual to individual). If the body temperature before irradiation is close to the fever point, it will climb beyond this point in the accumulator. Thus orgone energy can produce mild fever. Temperature rise in the organism is known to be a fundamental excitation reaction of the cells and the blood. Until now it has not been understood. …” [2]

“The energy fields of the two systems make contact and after some time, dependent of the bio-energetic strength of the organism within, both the living organism and the energy field of the accumulator begin to “luminate,” i.e., they become excited and, making contact, drive each other to higher levels of excitation. This fact becomes perceptible to the user of the orgone energy accumulator through feeling of stronger prickling, warmth, relaxation, reddening of the face, and, objectively, through increased body temperature (0.5 to 1.5 °F) [0.28 to 0.83 °C]. The body temperature should be measured before and during the irradiation (not after the sitting)” [3]

However, apart from these observations Reich never did systematic investigations and never published data regarding the variation of the body core temperature when an organism is exposed to concentrated orgone energy fields. After Reich’s death (1957) many researchers carried out investigations to study and evaluate this difference. Ritter and Ritter [4] observed an increase of body temperature when an organism was kept inside an ORAC. They carried out experiments on 9 patients who underwent 45 sessions inside an ORAC for a duration of 45 minutes each. They found an average increase in the body temperature of 0.48 °F (about 0.27 °C). Muschenich and Gebauer [5] carried out a double-blind experiment on 10 volunteers kept for 10 sessions of 30 minutes each inside an 8-fold ORAC. They also carried out an additional series of 10 sessions of 30 minutes each inside a control box with the same characteristics but without the metallic layers. They observed an average increase of the body core temperature after the sessions inside the ORAC of 0.21 °C, while the average increase of body core temperature for the sessions inside the control box was 0.04 °C. This being a significant difference statistically (P = 0.01). The American government body, the FDA (Food and Drug Administration) did an investigation on the medical effects of the ORAC through a professor of physical medicine at the Mayo Foundation. Twelve human subjects were tested on blood pressure, respiratory rate, temperature and pulse rate. The subjects sat in the ORAC for thirty minutes daily, six days a week for three consecutive weeks. The FDA surprisingly confirmed the patient’s temperature effects to be in line with that which Reich had claimed, with changes as high as 0.4 °F (0.22 °C) in some of the tests carried out. However, they spuriously dismissed this as, ‘not spectacular’ [6].

Snyder [7] carried out a single blind study designed to verify the possible effects of the ORAC on subjects sitting inside for 15 minutes. Finger temperature was one of the parameters monitored in the study. A two-fold ORAC was used in the study together with a control box. Thirty volunteers, 21 female (70%) and 9 males (30%) were enrolled in the study. Prior to the session, finger temperature baselines were established when temperatures did not fluctuate more than 0.2 °F (about 0.1 °C) in a one-minute period. Subjects were then randomly assigned to either the ORAC or the control box, but never to both. After a one-minute wait, finger temperature was taken at one-minute intervals for the next 15 minutes. A mean rise of 3.4 °F (about 1.9 °C) for the subjects exposed in the ORAC, as compared to a mean rise of 0.7 °F (0.4 °C) for the control subjects, was recorded. Using a one-way analysis of variance, the ORAC was significantly different from the control box (p<0.04).

Correa and Correa [8] carried out measurements of the oral temperature readings on a male patient (54 years old) before entering, and after 15 minutes treatment inside a 5-fold ORAC for 10 consecutive days. They observed in all sessions a positive variation of the temperature reading except for one day where the weather conditions were characterised by much cloud coverage. In all the other days of the study the sky was clear. The average temperature increase was 0.42 ± 0.05 °C.

More recently, Mazzocchi and Maglione [9] carried out measurements of body temperature on 20 healthy patients before and after 45-minute treatments inside a 5-fold ORAC. Sessions were undertaken in September and October 2008. The data indicated a statistically significant average increase in body temperature of 0.242 °C (p = 0.006 for a significance level of p < 0.05).

Recently, Maglione and Piergentili [10] carried out body temperature measurements on a male patient with no major diseases before and after 20-minute sessions inside a 5-fold ORAC. Sessions were done daily from April to December 2021 for a total of 226 sessions. A statistically significant average increase of the body temperature of 0.146 °C was found (p = 2.381∙10-37 for a significance level of p < 0.05). When the variation of body temperature was linked to the weather conditions they found the average increase of body temperature was higher in clear, scattered clouds, and windy days (151) with 0.153 °C than in cloudy, overcast, and rainy days (75) where the average increase of body temperature was 0.130 °C.

However, notwithstanding the large body of investigations carried out in the last decades on the variation of body core temperature no study has been done correlating this variation with changes in the bio-energetic level and overall health of the subject. Temperature data from daily exposure inside an ORAC and health conditions related to four volunteers were collected and analysed. In one case, a correlation between temperature difference and variation of the energetic charge of the blood, and the presence of Ca I-III cells in biological tissues was also considered and evaluated. Temperature data before and after daily sessions with a 3-fold ORAC were spontaneously and voluntarily provided to the author by the volunteers that routinely and continuously used the ORAC. Volunteers’ personal data were not available due to privacy protection.

Materials and Methods

Four volunteers carried out temperature measurements before and after each session inside the ORACs, which were made available at their homes. The characteristics of the ORACs were similar, being of the same sizes, and made of three alternated layers of sheep wool and steel wool. The volunteers recorded the temperature by axilla measurements using an analogic thermometer. The same axilla was used in all the measurements. The thermometer was zeroed after the measurement before entering the ORAC in such a way to make it available for the second temperature measurement to be done right after the conclusion of the session. Thermometers when not in use were kept outside the ORACs. The measurements were done always with the subjects outside the ORAC. The duration of each session ranged between 45 and 60 minutes for all volunteers. The treatment inside the ORAC’s was made with a daily frequency. In some cases more than one session was done.

In the present study the variation and the trend of the temperatures before and after a session was evaluated on a monthly basis. In order to evaluate the measured temperature data the following parameters had been developed and considered:

  1. charging capacity
  2. charging potency.

The charging capacity parameter expresses the ratio of the summation of all sessions with positive temperature difference to the total sessions done in the reference month. The unit of measurement is [%]. It is evident that if all the 31 (or 28-30) sessions done in the reference month show a positive temperature difference, the value of the charging capacity would 100 %, or:

In case, in the reference month, some sessions recorded a negative value of the temperature difference, the charging capacity will be less than 100 %. For example, if in the reference month, 6 temperature differences out of the 31 (or 28-30) were negative, the charging capacity will be given by:

In this case the charging capacity will be 80 %.

The charging potency indicates the recharge strength of the organism. It is represented by the average difference of the temperature recorded before and after the sessions done in the reference month. In other words, the charging potency is given by the algebraic summation of all the positive and negative temperature differences recorded in the sessions within the reference month. This figure would be divided by the number of sessions. The unit of measurement is [°C]. It is evident that the higher and positive the average temperature difference, determined in the reference month, the stronger the energetic recharging of the organism. For example, if 31 sessions are performed in a month and the algebraic summation of the temperature differences related to all the sessions is 12.0 °C, the charging potency will be 0.39 °C, or:

The above value indicates that in the reference month the patient observed in each session an average increase of body temperature of 0.39 °C. In other words, patient temperature after each session was higher than the temperature recorded before the session by 0.39 °C. If we assume that the energy charge of the organism is directly related to the increase of the body temperature, we can argue that the higher the value, the stronger the recharging of the organism.

Data and Results

The four volunteers participating in this study are referred to as subject A, B, C, and D. Subject A, a female, had a bowel tumour that was removed by surgery. After the removal, the subject was greatly debilitated and in precarious health conditions, notwithstanding the official therapies. She decided to start using daily and continuously the ORAC from December 2012 to August 2022, for a total period of 117 months or nearly 10 years. Behaviour of the charging capacity parameter and of the charging potency parameter versus time is reported in the graph in figure 1. The (horizontal) x-axis reports the time, while the (vertical) y-axis reports the two parameters. Red squares represent the behaviour of the charging potency, while blue squares the charging capacity. Each square for both trends represent the analysis of the temperatures before and after the ORAC’s sessions recorded in the month corresponding to the number on the x-axis. For example, 1 on the x-axis denotes the first month in which the ORAC had been used, 10 in the same axis the tenth month after the beginning of the use of the ORAC, etc.

Figure 1

From the trend of the charging capacity and of the charging potency, as reported in Figure 1, the bio-energetic condition of the subject can be deduced. Indeed, in the first six months of use of the ORAC the subject had a very low charging capacity with values much lower than 50 %. From the 5th month on, the trend started increasing and recording good values. From the 31st month on it stabilised at the value 1.0 (100 %). Equally interesting is the trend of the charging potency that for the first 6 months was very negative. A minimum was reached at the 4th month with -0.93 °C. Only from the 7th month on, a slow but progressive and continuous increasing trend of the temperature difference was observed and at the 73rd month reached a value of 2.0 °C. From the two trends it is evident that at the beginning of the use of the ORAC the subject clearly showed a very low energetic charge and a low orgonomic potential that kept her from charging and making the charging potency increase.

The behaviour and the evolution of the charging potency over time can be explained by reference to the law of the orgonomic potential. At the beginning, the energetic charge of the ORAC, evidently high, was in competition with the low energetic charge of the patient and, according to the law of the orgonomic potential, the orgone energy flowed mostly from the patient towards the inside of the ORAC (rather than the opposite way around in order to recharge the organism). This effect must be considered when using an ORAC with an energetically very weak organism. The use of a strong ORAC is highly discouraged if the aim is to recharge very weak organisms. A weak organism with a low content of orgone energy but not completely energetically drained, can increase its charge and hence its orgonomic potential by using adequate procedures, such as gradual use, increasing from low to higher charge devices, or reducing the daily exposure time. Undergoing daily half an hour session by using a 1-fold ORAC, or some 5–10-minute sessions by using a 3-fold ORAC might be suggested. When the organism has acquired a sufficiently charge, evidenced by an increasing trend of the charging capacity and charging potency parameters, one can then move to permanently using a 3-fold ORAC and follow the standard protocols. In a contrary situation, the organism might lose bioenergy and recharge capacity instead of acquiring it. This might be evidenced by a steadiness, or even by a decrease in the time of the charging capacity and charging potency parameters.

The slow and gradual increase of its orgonomic potential makes the organism increasingly stronger in respect to the ORAC which indeed has a charge that remains, within certain limits, practically constant against time. At this point the organism starts an energetic charging.

Subject B. The subject, a female, had a breast tumour. She used the ORAC daily and continuously from April 2019 to April 2022, for a total period of 37 months or just over 3 years. Behaviour of the charging capacity parameter and of the charging potency parameter versus time is reported in the graph of figure 2.

Figure 2

From the graph it can be seen the charging capacity for the 1st month was quite low and equal to 0.73 (73 %). By the daily use of the ORAC, the charging capacity soon showed an improvement and reached 100 % at the 2nd month, a value that remained constant in time. Similar is the charging potency behaviour that, with an increasingly recharged organism, increased its value. Charging potency in the 1st month is practically zero (0.02 °C) but is increasing harmoniously and progressively in the following months with a clear increasing trend.

It must be emphasised that from experience, the generalised improvements of a subject’s health conditions are usually perceived only after some months from the start of the use of the ORAC, with a concomitant improvement of the traditional clinical exams (markers, echography, CT scan, etc.). Generally, this might occur when the charging potency starts permanently exceeding the threshold of 0.4-0.5 °C, this for subject B was after the 19th–20th month.

Also, Subject B, for privacy, did not provide any clinical information that could confirm her better health conditions after the analysed period. To date, she is very satisfied and in net recovery.

Subject C. The subject, a male, lent himself to the present study with no major pathologies. So, any reaction to the use of the ORAC might be considered those of a healthy person. He used daily the ORAC for 37 months from April 2019 to April 2022.  Behaviour of the charging capacity parameter and of the charging potency parameter versus time is reported in the graph of figure 3.

Figure 3

As expected, the charging capacity showed a prompt response to the use of the ORAC and soon reached the value of 1.0 (100 %) that was maintained for the whole study period. The charging potency, instead, showed high fluctuations versus time. In fact, notwithstanding he was healthy, the initial charging potency was low and remained below 0.40 °C for three months. Then, it quickly reached a peak of 1.0 °C, to go down slightly in the following months. To date, no explanation is available for this behaviour. However, notwithstanding the fluctuation of the values, the charging potency showed an increasing trend versus time. Since he started using the ORAC, the subject observed increased and better general health conditions.  

Subject D. The subject, a female, did not have evident pathologies. She underwent the microscopic test (Reich Blood Test) and the cytologic test on urinary sediment early in 2021. The microscopic exams clearly showed a strong T-reaction of the red blood cells that were in most of them thorny in appearance, as showed in the following pictures (a) through (f) in Figure 4.

Figure 4

Although there was an abundant bacterial infection that obfuscated the slides, the cytologic test clearly showed cells at different stages of bionous disintegration as reported in the pictures of Figures 5 through 8 [11].

Figure 5
Figure 6
Figure 7
Figure 8

Figure 5 shows very bionously disintegrated Ca I cells, while images (a) through (d) in Figure 6 show clusters of Ca II cells. Images (a) through (d) in Figure 7 show single Ca II cells, and images (a) through (m) in Figure 8 indicate not completely mature Ca III cells.

After a first substantation of the low energetic status indicated by the Reich Blood Tests (microscopic and the cytologic test), Subject D decided to start using the ORAC as a tool for recharging himself bio-energetically. She used the ORAC daily for 8 months from October 2021 to May 2022. Behaviour of the charging capacity parameter and of the charging potency parameter versus time is reported in the graph of Figure 9.

Figure 9

From the above graph it can be observed that at the beginning both the charging potency and the charging capacity were very low. In fact, for 4 months both parameters did not reach acceptable values (being the charging potency is lower than 0.4-0.5 °C, and the charging capacity is lower than 1.0 or 100 %). However, it can be also noted that the daily use of the ORAC made the two parameters move almost in parallel towards an optimistic rising trend that might be considered a prelude to a bio-energetic recharging.   

After 8 months from the beginning of the daily use of the ORAC a second microscopic and cytologic test was done. The two tests highlighted improved conditions of the blood that was much better charged even though the situation at cellular level was found unchanged. Pictures in Figures 10 and 11 refer to the second exam. 

Figure 10
Figure 11

Images (a) through (f) in Figure 10 show a B-rection of the cells, while images (a) and (b) in Figure 11 still show Ca II cells. Images (c) through (h) continue to evidence the presence of Ca III cells. Clearly the biological tissues benefit most from the improved energetic charge of the blood in that they recharge first and more rapidly, particularly the red blood cells which after a few months show a higher charge.

As for the other biological tissues, only the Ca I cells find benefit from the increased bio-energetic charge of the organism. Ca I cells are those that first start the bionous disintegration. Due to the recharging process this disintegration no longer occurs and, in optimal conditions, the bions just produced are reabsorbed. Instead, the increase of the bio-energetic charge does not affect the existing Ca II and Ca III cells. Once formed, they continue to exist. However, no new Ca II and Ca III cells form since the recharging of the organism succeeds in stopping the bionous disintegration and thus the formation of new Ca I cells. Therefore, no new Ca II and Ca III cells can originate from further Ca I cells. In order to observe a change of the cytologic picture more than 8 months of use of the ORAC would be required. To get a confirmation of further improvements in a cytologic test one must wait till the next exam.


Presently, there are very few parameters to determine and evaluate the efficacy of an ORAC on the bio-energetic recharging of an organism when exposed to the concentrated orgone field. Amongst such parameters we find the increase of body core temperature. However, the efficiency of an ORAC might also depend on both the number of folds with which it is built, the organic and inorganic materials used and the energetic level and health conditions of the user.

Monitoring the charging capacity and the charging potency variables allows the researcher to observe and track the long-term effects of the ORAC. The trend of the two parameters provides useful indications. The charging capacity indicates whether a subject is recharging while the charging potency indicates how much the bio-energetic charge might be. In turn, these data points provide useful information on how the subjects react to the use of the ORAC and whether they take advantage from it. In some cases, because of the law of the orgonomic potential, where the stronger orgonotic system attracts orgone energy from the weaker orgonotic system, the recorded temperature data might indicate that the subject cannot recharge due to its energy being very low. For those subjects the use of weaker ORACs is required but sometimes a full recharging might still be problematic. Generally, this occurs with very weak subjects whose exposure dosing becomes very difficult. Terminal patients do not get appreaciable improvements and advantage from the use of an ORAC mainly for this reason. In some cases, the problem can be solved by using the same 3-fold ORAC and doing frequent daily sessions of a few minutes for several weeks and then monitoring the trend of the body temperatures. This procedure might allow a very energetically low subject to recharge by short and frequent doses of daily orgone energy, in a similar way to which a severely undernourished subject might be brought to normal by small and frequent meals based on tea, cookies, and soups.

Sometimes, we have witnessed real successes and, if the patient responds well to the use of the ORAC, after a few weeks the duration of the sessions can be increased in steps of 5 minutes daily towards a maximum duration of 45-60 minutes per session, twice a day. Obviously, if one increases the duration of the daily sessions, their frequency should be decreased.

There is a last point that emerges from the temperature monitoring and the trend of the charging capacity and charging potency parameters. These parameters parallel the improvement of a subject’s health conditions, the reversing of the cancer cells process and the disapppearing of the Ca I cells. They also correlate with the cessation of new Ca II and III cells (from Reich’s microscopic and cytologic tests). The overall point being that the cancer disease process might not be simply abscribable to the genetic traits of the subject alone. It is clear that the health’s conditions and Ca I cancer cells’ picture can be improved within a few months by bio-energetically recharging the organism with a systematic use of Reich’s ORAC. This result might call into question a solely genetic theory of the cancer disease process attributing its development rather to a decrease of the bioenergetic level of the organism.


Reich’s ORAC is an excellent tool to be used in cancer patients, mainly in the preventive phase. The increase of the bio-energetic level of the organism slows down or even stops the bionous disintegration of the cells. In turn, the lack of bions avoids the formation of those ‘bricks’ from which the cancer cell is made. In this way, the cancer process comes to a significant and subtantial halt. In the best cases, when a certain limit of the charging potency is exceeded, the subject is able to perceive subjective improvements of its bio-energetic charge, such as a feeling of general wellbeing, improvement of skin tone and a generic recovery of the strength. This is often concomitantly confirmed by objective data such as the results of traditional lab analysis that considerably improve.    

Another important aspect that emerges from the evaluation of the trend of the charging potency and charging capacity is how the bio-energetic charge of the organism controls the formation of the cancer cells. This goes some way in challenging the fundamentals of the genetic theory of the disease.


The author wishes to thank Roberto Maglione and Leon Southgate for their suggestions in writing the paper.


  1. T is the temperature measured outside the ORAC, and T0 the temperature inside the ORAC. 
  2. Reich W, The Cancer Biopathy, Orgone Institute Press, New York, 1948 page 317.
  3. Reich W, The Orgone Energy Accumulator, Orgone Institute Press, Rangeley, Maine, 1951, page 27. A temperature difference of 0.5 °F corresponds to about 0.3 °C; while 1.5 °F to about 0.8 °C.
  4. Ritter P, Ritter J, Experiments With The Orgone Accumulator, The Ritter Press, Nottingham, UK, undated (circa 1959).
  5. Muschenich S, Gebauer R, The Psycho-Physiological Effects Of The Reich Orgone Energy Accumulator, Pulse of the Planet, Natural Energy Works, Ashland, Usa, Vol. 1, N° 2, 1989; see also Muschenich S, Einige Zitate Und Bemerkungen Zur Frage ‘Sind Die Begriffe Vagotonie Versus Sympathocotonie Uuf Die Wirkungen Des Orgons übertragbar?’, Emotion, N° 8, pp. 68-71, 1987; and Müschenich S, Der Gesundheitsbegriff im Werk Des Arztes Wilhelm Reich (The Concept Of Health In The Works Of Dr. Wilhelm Reich), Doktorarbeit am Fachbereich Humanmedizin der Philipps-Universitat Marburg, Verlag Gorich & Weiershauser, Marburg, 1995.
  6. Blasband RA, An Analysis Of The U.S. Food and Drug Administration’s Scientific Evidence Against Wilhelm Reich, Journal of Orgonomy, Vol. 6, No. 2, November 1972, pages 208-210.
  7. Snyder NR, Finger Temperature Effects Of The Orgone Accumulator, Journal of Orgonomy, 23(1):57-63, 1989.
  8. Correa PN, Correa A, Transiently Induced Hyperthermia In Humans Exposed To A Controlled Orac Environment, ABRI Monographs: Biophysics Research AS2-33, Toronto, Canada,  2006.
  9. Mazzocchi A, Maglione R, A Preliminary Study Of The Reich Orgone Accumulator Effects On Human Physiology, Subtle Energies & Energy Medicine, Vol 21, N° 2, 2010.
  10. Maglione R, Piergentili L, Temperature And Electric Measurements On An Organism Exposed To A Concentrated Orgone Energy Field, Journal of Psychiatric Orgone Therapy, May 02, 2022.
  11. Bions are minute pre-cellular vesicles that Reich discovered can be created within the body or outside of it through disintegration and reorganisation processes.

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A Pictorial Essay of Photographic Evidence of Healthy and Cancer (Phase I) Cells, and RBCs After the Reich Biological Blood Tests

Editor’s Notes:

This pictorial essay is an important step in introducing the concept of cancer biopathy to contemporary medicine.

The following paragraph written by Dr. Wilhelm Reich in the book "Cancer Biopathy" reflects the significance of this article written by Armando Vecchietti.

"The examination of the blood is therefore particularly useful for the early detection of cancer. In fact, I would like to venture assumption that the blood is the first system to be affected by systemic contraction and subsequent shrinking of the organism. Blood is, after all the "sap of life" which binds all the organs into one whole and provides them with nourishment. Blood therefore plays a major role in orgone therapy for cancer. For that reason, the orgonothic function of the blood must be fully understood."  Reich W. Cancer Biopathy (page 235).



The paper reports a collection of photographs regarding the behaviour of healthy and phase I cancer process (Ca I, Ca II, and Ca III) cells. Figures describing the behaviour of energetically-charged or weak RBCs (Red Blood Cells), when made to disintegrate in saline, and then subjected to autoclavation are also reported. The compendium fills a gap within orgonomic medicine, and might be considered as an important reference for further studies in the evaluation of healthy and cancer conditions of the human organism.


When I began to study orgonomy in the mid-1970s and specifically the Reich blood test for the very early diagnosis of cancer I soon realised that an exhaustive amount of photographic material to be considered as reference was not available.

Images of the cancer cells and of the RBCs as described by Reich practically did not exist, and only a few drawings and some black and white pictures were available.1

After Reich’s death very few articles were published reporting high-quality images of the cancer cell’s formation and process, and very little regarding those cells characterising the first phase (phase 1) of the cancer process (Ca I, Ca II, and Ca III cells), and about the results of the Reich biological blood tests.2

Therefore, being that the amount of published material was far from exhaustive, I could not compare what I saw at the microscope with other material because of its low quality or even the lack of it altogether. Many years were needed before much light was shed on the interpretation of several images I had personally produced.

The following collection of photographs has the purpose to fill this gap and to offer images the reader can use as reference in future work thus making easier ongoing study and research. The photographs show images of disintegration of urinary tract cells and RBCs. The various form and shapes they assume is of course a function of their orgonotic or energetic charge. Both groups of cells had been taken from samples from human beings of both sex and of different ages. All the biological samples had been observed at the optic microscope and photographed in-vivo with a magnification ranging between 800x and 1000x.3 All images had been developed in-house and belong to the archives I built in more than 40 years of research.

As to the images regarding the Ca I, Ca II, and Ca III cells, the photographed material comes from centrifuged urinary sediments only as I consider this the easiest to obtain and observe amongst the various other sources available, such as the patient’s sputum.

As to Ca IV (mature) and Ca V (putrid decomposition) cells belonging to phase 2 of the cancer process, no images have been reported in this present collection since they are well-known from official oncology and a large selection is available in the mainstream scientific literature. The reader can refer to that for reference.

Images regarding the blood are instead of RBCs only. For them the distinction is simpler because we can observe only two different behaviours:

  1. The B-reaction, an expression of the good energetic charge of the RBC, characterised by the presence of large bluish bions embedded in the stroma of the cell like pearls in a ring.4
  2. The T-reaction, an expression of a low energetic charge of the RBC, characterised by a contracted and thorny (or T-spikes) cell.5

As far as the pictures taken at the autoclavation test is concerned they refer only to the macroscopic aspect of the autoclaved blood samples, and how they appear at the end of the autoclavation test in keeping with their energetic charge.

The present collection of images starts with a brief overview of healthy cells. This compendium might be useful to scholars to realise what might be the standard aspect of energetically-charged cells so as to have a baseline when pathological variations, such as those occurring in the Ca I, Ca II, and Ca III steps, appear over time. A comprehensive compendium of the latter is reported thereafter. Finally, a brief presentation of some examples of RBCs from the Reich blood test and of blood samples after the autoclavation test, is also reported. 

Healthy Cells

Figures 1 through 4 in this section show healthy or energetically-charged cells. The images were taken by a 800x-1000x microscope. Only a membrane that contains a substance called cytoplasm in which the cell nucleus floats can be clearly observed.

Figure 1 – Healthy or energetically-charged cell
Figure 2 – Healthy or energetically-charged cells
Figure 3 – Healthy or energetically-charged cells
Figure 4 – Healthy or energetically-charged cells

Ca I cells

The Ca I cells are those that show bionous disintegration. Reich explained how the bionous disintegration is directly activated by the action of the T-bacilli on the weakest cells of the tissues.6 In the previous section of healthy cells of a human organism, observed in-vivo at the optic microscope, it could be seen to show a clear cytoplasm. The Ca I cells instead appear granulated because of the presence of vesicles (bions) and/or tiny T-bacilli.

In this first step the affected cells generally tend to get rounder because of contraction. In the epithelial cells this contraction can be clearly seen in the corners of the cell that become more and more rounded making the cell gradually lose its pentagonal shape.

When the Ca I cells follow also a T-reaction (thorny) of the RBC, a diagnosis of Ca I step of the cancer process might be done.

This is the first step of the cancer process and the organism starts to move forward along a cancer pathology. This first step, as well as the successive steps Ca II and Ca III are still unknown to classic oncology. It does not understand their meanings being not able to interpret the above-mentioned behaviours.

Following Figures 5 through 22 show examples of Ca I cells. Figure 17 shows an example of a set of healthy cells and cells that are about to disintegrate.

Figure 5 – Ca I cells
Figure 6 – Ca I cell
Figure 7 – Ca I cell
Figure 8 – Ca I cells
Figure 9 – Ca I cell
Figure 10 – Ca I cells
Figure 11 – Ca I cell
Figure 12 – Ca I cell
Figure 13 – Ca I cells
Figure 14 – Ca I cells
Figure 15 – Ca I cells
Figure 16 – Ca I cells
Figure 17 – Healthy and Ca I cells
Figure 18 – Ca I cells
Figure 19 – Ca I cell
Figure 20 – Ca I cells
Figure 21 – Ca I cell
Figure 22 – Ca I cell

Ca II cells

In this second step of the phase 1 process bions (or vesicles) start to aggregate and the energy concentrates itself in the bionous clusters both inside and outside the cells. These bionous clusters reorganise themselves by producing a cellular membrane that wraps them and evolves into structures that sometimes develop at the expense of the old cell.

Both inside and outside the affected cells the presence of new varyingly defined tapered or ovoid shapes can be observed. In some cases the remaining part of the affected cell breaks up into small fragments, small vesicles or T-bacilli. The arrows in some of the following images show the formation of new structures that are the result of the previous fusion of bions clusters. The whole is a Ca II cell.

In this step again, classic oncology does not recognise the presence of cancer cells, and hence to it no cancer pathology is attached.

Following Figures 23 through 57 show examples of Ca II cells.

Figure 23 – Ca II cells
Figure 24 – Ca II cell
Figure 25 – Ca II cells
Figure 26 – Ca II cells
Figure 27 – Ca II cell
Figure 28 – Ca II cell
Figure 29 – Ca II cell
Figure 30 – Ca II cell
Figure 31 – Ca II cells
Figure 32 – Ca II cells
Figure 33 – Ca II cells
Figure 34 – Ca II cell
Figure 35 – Ca II cell
Figure 36 – Ca II cells
Figure 37 – Ca II cell
Figure 38 – Ca II cells
Figure 39 – Ca II cells
Figure 40 – Ca II cells
Figure 41 – Ca II cells
Figure 42 – Ca II cells
Figure 43 – Ca II cells
Figure 44 – Ca II cell
Figure 45 – Ca II cells
Figure 46 – Ca II cell
Figure 47 – Ca II cells
Figure 48 – Ca II cells
Figure 49 – Ca II cells
Figure 50 – Ca II cell
Figure 51 – Ca II cell
Figure 52 – Ca II cell
Figure 53 – Ca II cells
Figure 54 – Ca II cells
Figure 55 – Ca II cell
Figure 56 – Ca II cells
Figure 57 – Ca II cells

Ca III cells

Once the Ca II cells have been formed they continue to evolve and, in their progressive development they rearrange and give rise to the Ca III cells. The Ca III cells are the last step of the evolutionary stage of the clusters (phase 1). They are called also club-shaped or caudate cells and are the prelude to the tumour mass (phase 2 of the cancer process). They are cells completely new and foreign to the organism in that they do not belong to any human tissue.

Following figures 58 through 120 show examples of Ca III cells.

Figure 58 – Ca III cell
Figure 59 – Ca III cell
Figure 60 – Ca III cell
Figure 61 – Ca III cells
Figure 62 – Ca III cell
Figure 63 – Ca III cell
Figure 64 – Ca III cell
Figure 65 – Ca III cells
Figure 66 – Ca III cell
Figure 67 – Ca III cells
Figure 68 – Ca III cell
Figure 69 – Ca III cell
Figure 70 – Ca III cell
Figure 71 – Ca III cell
Figure 72 – Ca III cell
Figure 73 – Ca III cell
Figure 74 – Ca III cell
Figure 75 – Ca III cells
Figure 76 – Ca III cell
Figure 77 – Ca III cell
Figure 78 – Ca III cells
Figure 79 – Ca III cell
Figure 80 – Ca III cells
Figure 81 – Ca III cell
Figure 82 – Ca III cell
Figure 83 – Ca III cell
Figure 84 – Ca III cell
Figure 85 – Ca III cells
Figure 86 – Ca III cell
Figure 87 – Ca III cell
Figure 88 – Ca III cell
Figure 89 – Ca III cell
Figure 90 – Ca III cell
Figure 91 – Ca III cell
Figure 92 – Ca III cell
Figure 93 – Ca III cell
Figure 94 – Ca III cell
Figure 95 – Ca III cells
Figure 96 – Ca III cells
Figure 97 – Ca III cell
Figure 98 – Ca III cell
Figure 99 – Ca III cell
Figure 100 – Ca III cell
Figure 101 – Ca III cell
Figure 102 – Ca III cell
Figure 103 – Ca III cell
Figure 104 – Ca III cell
Figure 105 – Ca III cell
Figure 106 – Ca III cell
Figure 107 – Ca III cell
Figure 108 – Ca III cell
Figure 109 – Ca III cells
Figure 110 – Ca III cell
Figure 111 – Ca III cells
Figure 112 – Ca II (above) and Ca III (below) cells
Figure 113 – Ca III cell
Figure 114 – Ca III cells
Figure 115 – Ca III cell
Figure 116 – Ca III cells
Figure 117 – Ca III cells
Figure 118 – Ca III cell
Figure 119 – Ca III cell
Figure 120 – Ca III cell

The Reich Blood Test

The test is a method for determining the status of a patient’s health, and the onset and progress of a cancerous process at work within the organism. It focuses on the different response of the RBCs when made to disintegrate in physiological salt solution. The cells show two different reactions according to the orgonotic charge they possess. In case the RBC presents a strong orgonotic charge, it is taut and shows a strong and well delineated membrane, the stroma is filled by bluish vesicles that look like pearls set in a ring. Conversely, if the RBC possesses a low orgonotic charge, the cell is energetically weak and the volume of the stroma gets smaller, somewhat shrunken, and has a thorny appearance like the hedgehog of a chestnut, or a medieval spherical flail covered in spikes. Reich called this latter configuration a T-spikes cell.

The overall evaluation of the energetic charge of the RBCs in a blood sample depends on how many of them are energetically strong (vesicles set like pearls in a ring) and how many are energetically weak (thorny appearance). As a consequence, the overall orgonotic charge may vary between the two above extremes with all the possible combinations in between.

Figures 121 through 124 show examples of a B-reaction of the blood (energetically strong RBCs); while in figures 125 through 127 examples of blood samples characterized by a T-reaction (energetically weak RBCs) are seen.

Figure 121 – B-reaction of the blood
Figure 122 – B-reaction of the blood
Figure 123 – B-reaction of the blood
Figure 124 – B-reaction of the blood
Figure 125 – T-reaction of the blood
Figure 126 – T-reaction of the blood
Figure 127 – T-reaction of the blood

The autoclavation test

The autoclavation test is a cohesion-type test based on an assumption that healthy RBCs withstand the autoclavation better than cells with a low orgonotic charge. Energetically-charged blood after the autoclave appears as a compact agglomeration surrounded by a clear supernatant liquid. An energetically weak blood shows a turbid fluid in which a various degree of fraying can be observed. In the worst cases the appearance of the fluid might reach that of a greenish murky mush. As observed for the blood test, the result obtained by the autoclavation test is never characterised by a single condition. Rather, many intermediate forms between the two extremes can be found. A correct evaluation might be done only by a well-trained and expert orgonomist.

Figures 128 through 130 show examples of energetically-charged bloods where a dense clumping can be seen, while picture in figure 131 reports two vials showing how it appears an energetically-weak blood after the autoclavation test.

Figure 128 – Energetically-charged blood
Figure 129 – Energetically-charged blood
Figure 130 – Energetically-charged blood
Figure 131 – Energetically-weak bloods


The author wishes to thank Roberto Maglione and Leon Southgate for their suggestions in writing the paper.


1 The only available material graphically or pictorially describing the cancer cell formation Reich made public was contained in the book The Cancer Biopathy. Volume II of the Discovery of the Orgone, Farrar, Straus and Giroux, New York, 1973; and in the following articles: 1) The Natural Organization of Protozoa from Orgone Energy Vesicles (Bions), International Journal of Sex-Economy and Orgone-Research, Vol 1, N° 3, Orgone Institute Press, New York, November 1942; 2) Experimental Orgone Therapy of the Cancer Biopathy (1937-1943), International Journal of Sex-Economy and Orgone-Research, Vol 2, Orgone Institute Press, New York, 1943; 3) “Cancer Cells” in Experiment XX, Orgone Energy Bulletin, Vol 3, N° 1, Orgone Institute Press, Orgonon, January 1951; and 4) Orgonomic Diagnosis of Cancer Biopathy, Orgone Energy Bulletin, Vol IV, N° 2, Orgone Institute Press, Orgonon, April 1952 (paper compiled by Raphael CM and MacDonald HE based on a course on cancer given by Reich at Orgonon in July and August 1950). As to the biological blood tests that included the Reich blood test, originally called by Reich T-blood test, and the autoclavation test, called by Reich also biological resistance test, information could be found scattered in the above papers even though drawing and pictorial material was rarely reported. It can be found especially in 4).

2 Lassek H, Gierlinger M, Blutdiagnostik und Bion-Forschung Nach Wilhelm Reich. Teil 1, Emotion, Berlin, May 1984; Cantwell AR, Blasband R, Bionous Tissue Disintegration in Three Patients with AIDS, Journal of Orgonomy, Vol 22, N° 2, November 1988; Cantwell AR, Bionous Disintegration in Degenerative Disease, Journal of Orgonomy, Vol 25, N° 2, November 1991; DeMeo J, The Biophysical Discoveries of Wilhelm Reich, Pulse of the Planet #4, Natural Energy Works, Ashland, 1993; Blasband RA, Transformationen in Mikrobiologischen Organismen, in DeMeo J, Senf B (Ed) Nach Reich. Neue Forschungen zur Orgonomie, Zweitausendeins, Frankfurt, 1997; DeMeo J, Bion-Biogenesis Research and Seminars at OBRL: Progress Report, Pulse of the Planet #5, Natural Energy Works, Ashland, 2002; and Reich W, Bion Experiments on the Cancer Problem, Abstract of a Lecture Given to the Norwegian Society of Medical Students in Oslo, June 1938, Orgonomic Functionalism, Volume 7, Spring 2019, Wilhelm Reich Infant Trust, Rangeley, Usa. As to the biological blood tests drawing and pictorial material can be found in Bradbury P, Blue Armour and the Reich Blood Tests, Energy and Character, Vol 4, N° 3, September 1973; Baker CF, Dew RA, Ganz M, Lance L, The Reich Blood Test, Journal of Orgonomy, Vol 15, N° 2, November 1981; Lassek H, Gierlinger M, Blutdiagnostik und Bion-Forschung Nach Wilhelm Reich. Teil 1, Emotion, Berlin, May 1984; Lappert PW, Primary Bions Through Superimposition at Elevated Temperature and Pressure, Journal of Orgonomy, Vol 19, N° 1, May 1985; Bauer I, Erythrocyte Sedimentation: A New Parameter for the Measurement of Energetic Vitality, Annals of the Institute for the Orgonomic Science, Vol 4, September 1987; Opfermann-Fuckert D, Berichte Uber Behandlungen Mit Orgonenergie, Emotion, Berlin, Vol 8, 1987; Opfermann-Fuckert D, Reports on Treatments with Orgone Energy, Annals of the Institute for the Orgonomic Science, Vol 6, September 1989; Baker CR, Burlingame PS, The Reich Blood Test, Annals of the Institute for the Orgonomic Science, Vol 6, September 1989; Blasband RA, Cappella R, Crist PA, Dunlap S, Foglia A, Konia C, Reich E, Schleining J, Radiation Victims and the Reich Blood Test, Journal of Orgonomy, Vol 24, N° 1, May 1990; Frigola C, Castro P, The Reich Blood Test and Autoflorescence, Journal of Orgonomy, Vol 25, N° 2, November 1991; DeMeo J, The Biophysical Discoveries of Wilhelm Reich, Pulse of the Planet #4, Natural Energy Works, Ashland, Usa, 1993; Blasband RA, Transformationen in Mikrobiologischen Organismen, in DeMeo J, Senf B (Ed) Nach Reich. Neue Forschungen zur Orgonomie, Zweitausendeins, Frankfurt, 1997; and DeMeo J, Bion-Biogenesis Research and Seminars at OBRL: Progress Report, Pulse of the Planet #5, Natural Energy Works, Ashland, Usa, 2002.

3 An optic microscope with an incorporated camera or video camera was used. In-vivo microscope examinations of the biological samples were performed by an Optika binocular optical microscope with 10x-20x-40x-100x objectives, and 15x eyepiece. A Panasonic NV-GS50 digital video camera, equipped with timer, was used in the recording of the microscope examinations.

4 The bions are primordial life forms-transitional structures between inorganic, non-motile forms and living, moving creatures capable of being cultured. They are preliminary stages of life and not completely forms of life. They can form from whatever substance be it human or animal tissue, earth, coal, moss, etc. (see Reich W, The Bion Experiments on the Origin of Life, Farrar, Straus and Giroux, New York, 1978).

5 The T-reaction is named by Reich after ‘Tod’ the German for death.

6 Reich W, Orgonomic Diagnosis of Cancer Biopathy, Orgone Energy Bulletin, Vol IV, N° 2, Orgone Institute Press, Orgonon, April 1952 (paper compiled by Raphael CM and MacDonald HE based on a course on cancer given by Reich at Orgonon in July and August 1950).

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Armando Vecchietti, MBiol

The paper reports and discusses a test for early cancer diagnosis. The test was developed by Reich and is based on his research on cancer biopathy, and on a new way to understand cancer mechanisms and processes. It can diagnose cancer greatly in advance of what the traditional tests can do and in addition in a period where no clues to the disease are supposed to exist by traditional oncology. It is focused on microscopic examination of biological samples such as blood, urine or sputum. Results of microscope laboratory examinations performed in-house on different biological samples, taken from patients with different health conditions, are shown and discussed in the paper. All the above materials belong to the laboratory’s archive of the writer.

The results fully confirm Reich’s assumptions and findings and evidence an early onset of the cancer disease even in people that are considered healthy and with no indication of the disease by classical oncology. In addition, it was observed that the development process of the precursors of the cancer cells (Ca I, Ca II, Ca III cells and T-bacilli) is common in humans and animals and it is irrespective of sex, age, and type of oncological pathology, being a universal process.

Introduction and background
Reich was the first to study in-vivo the behavior of living cells and tissues from a bioenergetic perspective by extensively investigating samples taken from healthy and cancer patients. He found that the bio-energetically weaker cells of the organism undergo, before the occurrence of the actual cancer disease as we characterize it today, slow but progressive changes that lead them finally to develop into the future cancer cell (1).

Reich found that the first phase in the formation of the cancer cell is characterized by indicators or precursors, such as bionous disintegration of the healthy cell and the associated development of the T-bacilli.
First studies date back to the late 1930s when he was in Oslo:

"3. Infusions of dried moss or grass collected in autumn show, if observed continually with a magnification of from 400x to 4000x, the following sequence of events: vesicular disintegration, after soaking and swelling separation of the vesicles in the form of cocci, concentration and organization (via formation of a membranous margin) into unicellular protists of vesicular structure ….

4. Exactly as protists develop out of swelled disintegrated moss, the cancer cells organize themselves in vesicular disintegrated animal and human tissues. The most important steps of this development are according to observations until now: Swelling of the tissue, vesicular structuring, formation of spindle-shaped organisms, strongly light-refracting and biologically stainable heaps of vesicles, and finally further development into mobile ameboid, creeping "mature" cancer cells." (2)

"These organisms [T-bacilli] result from degeneration of certain types of bion cultures, and from experimental degeneration of certain proteins. They are called T, i.e. "Tod" (Death) bacilli because of their origin and their deadly effect on mice. They are short flitting rods of about 0.25-0.6 μm. …. They have a sharp acid and ammonia-like smell. In large doses they kill mice within 24 hours. In small doses they produce in the course of from 8 to 15 months infiltrating and destructive growths in the form of tumors, or only cellular growths without a circumscribed primary tumor in kidneys, lungs, liver, glands, etc. They can be cultivated in pure culture from the cardiac blood of mice which died or were killed, and if again inoculated, they produce the same pathological changes as before." (3)

The identification, observation and study of the bionous disintegration and the formation of T-bacilli led Reich to understand the mechanism of the formation of the cancer cell and to provide a tool to make a very early diagnosis of cancer in a patient. This is in sharp contrast with classical oncology methodologies that require, in order to make a reliable cancer diagnosis, the cancer cell or the tumor to be already present in the patient.
Reich found the cancer cell formed and developed according to the following five different steps:

  1. Vesicular disintegration of cells (Ca I)
  2. Aggregation and reorganization of vesicles (Ca II)
  3. Evolution and development of caudate cells (Ca III)
  4. The mature cancer cell (Ca IV)
  5. The final phase of the cancer tumor: putrid disintegration (Ca V)

He called the cell characterizing each of the above steps Ca I, Ca II, Ca III, Ca IV, and Ca V respectively.

Each step is characterized by a specific cell shape and configuration. The full cancer process can be represented by two main distinct phases. Phase 1 is consisting of the steps Ca I, Ca II, and Ca III that Reich considered precursors of the future cancer cell and tumor mass as we know it today by classical oncology. While, phase 2 is characterized by the steps Ca IV and Ca V, where the cancer cell and the tumor mass are at this point well developed. It is noteworthy that the presence of Ca I, Ca II and Ca III cells allow us an early diagnosis in a period when, according to classical oncology, the disease does not exist yet.

Reich found this process common to all solid tumors and evidence that all types of tumor are characterized by the same mechanisms that at last lead to the formation of the characteristic cancer cell. In particular Ca I, Ca II, Ca III cells are the precursors of all solid tumor cancer cells and the different cancers are not different pathologies but the same pathology characterized by the same manifestations. In addition, Reich found that this process is typical of a human organism, without distinction of sex and age, and also of animals.

It is clear from the above classification that the working area of classic oncology is represented only by phase 2 of the full cancer process (Ca IV and Ca V steps), where the cancer cell and the tumor mass is present and developed at different degree. While, in phase 1 there is no recognition of potential indicators or precursors of a future insurgence of the cancer cell and disease.

According to these findings, Reich could develop a powerful tool for an extremely early diagnosis of cancer and determine, much in advance of the insurgence of the future cancer cell, all the concurrent conditions that could then give rise to the formation of the classical cancer cell. He observed that the blood played a key role in the early diagnosis process as, by circulating throughout the body, it might provide precious and first-hand information about the general energy condition of the organism.

The main characteristics and peculiarities of the steps grouped in phase 1 and 2 of the cancer cell formation, and development are briefly summarized in Figure 1.

Cancer consists of two phases. The 1st phase, completely unknown to classical oncology, consists of the transformation of healthy cells into the precursors of the cancer cells. While in the 2nd phase it is characterized by death and putrid decomposition of the mature cancer cells. The early diagnosis test identifies the transformations that occur in the 1st phase characterized by the presence of particular cellular formations that Reich called cells: Ca I – Ca II – Ca III.

The identification of these precursors, which appear much before the tumor mass or the mature cancer cells, allows the making of a very early diagnosis of cancer that anticipates by many years the common tests used today by classical oncology.

Figure 1 – Summary of the Cancer Process

Ca I cell. In this first step of the cancer process the low energetic level of the organism may affect the energetic qualities and characteristics of blood and tissues. Peculiar and meaningful alterations of the basic health conditions of blood red cells and tissues can be evaluated at the optical microscope. As far as the tissues are concerned, the tendency to a rapid vesicular reaction and the presence of T-bacilli are one of the most important indicators of the impending development of the cancer cell. As far as the blood red cells are concerned they show the two following features:

  • When they are energetically charged, they are visible at the microscope as bright, bluish spheres embedded like pearls in the stroma. Figure 2 shows an example of energetically charged red blood cells of a human blood examined at the microscope (picture from writer’s lab archive).
Figure 2
  • When their energy is very low, the red blood cells shrink and form thorns like chestnuts in their husk. Figure 3 shows an example of energetically weak red blood cells of a human blood examined at the microscope (picture from writer’s lab archive). This condition takes place much earlier than the formation of the first classical cancer cell, and is the very first indication of the potential formation of the future cancer cell.
Figure 3

Ca II cell. This step is characterized by an acute inflammatory condition. The vesicular reaction of the cells continues and evolves while the T-bacilli do not change and maintain the same characteristics throughout the cancerous process. The T-bacilli continue to trigger the vesicular reaction of the cells. The Ca II step is characterized by the fact the vesicles are starting to aggregate inside the cell along the cell membrane. The aggregations lose their vesicular feature to form a new structure that develops at the expenses of the original cell. Figure 4 shows an example of regrouping of bions inside a cell from a human urinary sediment (picture from writer’s lab archive). The arrow in the figure shows the vesicles that are starting to aggregate and merging.

Figure 4

Ca III cell. This step is characterized by a variety of features. The cells can appear spindle-shaped or club-tailed, with caudate, oval or round shape. Figure 5 shows an example of Ca III cell from a human urinary sediment (picture from writer’s lab archive). They also show a large variety of sizes, and an extraordinary variety of natural colors, in contrast with samples of artificially-colored tissues. They reproduce quickly as can be observed at the microscope.

Figure 5

All the cells above described generally have a low motility and are not too dangerous. However, the Ca III diagnosis contemplates a cluster of club-shaped cells that then are going to create the first tumor mass. This is the turning point of the early diagnosis. These cells are the last stage of early diagnosis.

With the presence of the Ca III cells, the formation of the tumor mass starts which only now becomes visible and recognizable and therefore can be diagnosed by classical oncology.

Reich found that the Ca III step was the most critical in the whole process, in that:

  1. patients that have Ca III cells but do not still have materialist evidence of cancer will develop the disease in the following period (months or years)
  2. all cancer patients show in their biological samples Ca III cells
  3. Ca III cells do not belong to any of human tissue.

The cancer process continues with two other steps, Ca IV and Ca V. Actually, we can speak of very early cancer diagnosis test only for Ca I – Ca II – Ca III cells which are characteristic of phase I and present much earlier than the mature cancer cells. With the formation of the tumor mass, however, the disease enters phase II and at this stage the cancer can be easily identified and diagnosed even with the normal tests currently used by classical oncology.
What follows (phase II with the Ca IV and Ca V steps) therefore does not fall within the frame that deals with the very early diagnosis but helps to briefly complete the picture and the description of the cancer process as a whole.

Ca IV cell. The formation of cancer cells and the tumor mass marks the beginning of phase II. In this phase the cells can have a round and elongated shape or become mobile due to the formation of pseudopodia which can sometime be filamentous.

In some cases, the vesicular aggregations develop a membrane that surrounds and envelops them thus developing a protozoa. The mobile protozoa is typical of the Ca IV step. Traditional oncology has repeatedly noted this protozoa but considers it just a parasite. If the body does not die sooner, these formations would become amoebae. At this stage, the malignancy of cancer depends on the degree of maturity of the cancer cells and on the speed at which the tissue is destroyed and decomposed.

Ca V cell. The Ca V step is characterized by necrosis namely a deadly and putrid decomposition of the cells of the tissues. It is the terminal phase of the cancer disease. When very mobile, tailed cells are found the cancer is very advanced. Microscopic image of the tissue in the Ca V step shows the presence of many debris, fragments of cells, vesicles, bacteria and, to higher magnifications, the T-bacilli. The Ca V step is determined and characterized almost exclusively by dying cancer cells that reached the end of their short lifecycle. While the body is still alive the decomposition of the tissues is comparable to the after-death necrosis. It results in a bacteremia and a generalized toxemia of the body. When the cancer does not affect vital functions, the death occurs by generalized putrefaction. This explains why, at last, the disease usually worsens rapidly into death. At this stage, any therapy is unsuccessful.

T-bacilli is a name coined by Reich from the German tod that stands for death, to recall the exceptional degree of danger these micro-organisms take once present and circulating in the living organisms. Cancer research has repeatedly noticed them but has always regarded them as an infection resulting from cancer and never understood their significance. T-bacilli are not new germs. Their most important biological characteristic is to attack the energetically weaker cells and stimulate their vesicular reaction. Indeed, T-bacilli attack healthy but energetically weak cells, forcing them to disintegrate vascularly. In this way, they close the circle of the cancer process triggering the formation of new Ca I cells. The presence of T-bacilli can be detected at the microscope with the darkfield technique on whatever cell, fluid or tumoral mass is obtained. Fresh material for fixing and staining that contains a great amount of T-bacilli can be easily aspirated from the center of a tumoral mass where the tissue is more decomposed.

Samples of very advanced tumor tissue, fixed and stained with hematoxylin and eosin and observed under the microscope, show large areas in the center of the tumor filled with T-bacilli in the form of tiny red dots. T-bacilli are Gram-negative (red-stained), with size of 0.2-0.5 μm. In comparison, vesicles (bions) are Gram-positive and blue-stained, with sizes of few μm.

In terminally-ill patients or in culture they produce a stink typical of an organism in putrefaction. In darkfield they appear as tiny lighted dots and when alive and active they show very fast zig-zag movements. They circulate freely inside the blood flow and in such a way they can reach and hit any cell in whatever part of the body. T-bacilli cannot be obtained directly from the air. Figure 6 shows T-bacilli observed at 400x darkfield microscope (picture from writer’s lab archive).

Figure 6

Materials and Methods
Over the last 30 years a large variety of biological samples were taken by the author from different patients with no clue of the cancer disease or with the cancer already present and developed at different degree and examined at the microscope. All cancer patients followed a course of treatment as prescribed by public or private hospital oncologists. The most taken samples were urinary sediment, blood and sputum. The biological samples were subjected to an in-house microscopic examination being the most suitable technique for this test.

An optic microscope (magnification between 400x and 800x) with an incorporated camera or video camera was used. The biological sample under testing was located on a slide and tested in-vivo as soon as possible. Microscope examinations were performed by an Optika binocular optical microscope with 10x-20x-40x-100x objectives, and 15x eyepiece. A Panasonic NV-GS50 digital video camera, equipped with timer, was used in the recording of the microscope examinations.

Results and Discussion
In this section images from different biological samples examined in-house by the author at the optical microscope are reported for each stage of the cancer cell formation and development process.

Figures 7 shows a healthy reaction of the red blood cells of an energetically healthy and charged patient.

Figure 7

Pictures in Figure 8 shows a magnification of healthy red blood cells.

Figure 8

The step 1 of the cancer process (Ca I) is represented by red blood cells with thorns similar to chestnuts. The cells are characterized by a low degree of bioenergy.  The Figures 9 and 10 show red blood cells from a bio-energetically weak organism and blood.

In the 400x magnified image (Figure 10) the typical appearance of low bioenergy red blood cells characterized by thorns similar to chestnuts in their husk can be observed.

Figure 9
Figure 10

When this framework occurs, and is associated to a pronounced spiky appearance of the red blood cells, a very first diagnosis of Ca I cancer cell (step 1) can be done. For classical Oncology no cancer is present at this time because no cancer cell is detectable yet.

The following Figures 11 and 12 show Ca I cells. These are the first cells that, losing energy, start the vesicular disintegration and produce vesicles.

Figure 11
Figure 12

The following Figure 13 and 14 show the two extreme limits of the red blood cells behavior. In Figure 13 all the red blood cells are energetically-charged, while in Figure 14 they are highly energetically-weak. However, in the daily-practice a limit situation alone can be rarely detected. Most often, an intermediate picture between the two above extreme conditions, where charged and weak red blood cells coexist in different percentages, is found.

Figure 13
Figure 14

If the red blood cells can display a smooth or thorny appearance, the vesicles formed in the Ca I cells are subjected to substantial modifications. They merge and aggregate themselves as shown by the arrows in Figures 15 through 18 giving rise to the formation of Ca II cells obtained from microscopic examination of a whatever vascularly disintegrated cells or from tissues of a bioenergetically-weak organism.

Particularly, in Figure 18, a vesicular aggregation, and evolution with the destruction of the original cell, can be observed.

Figure 15
Figure 16
Figure 17
Figure 18

The step Ca III is characterized by the presence of club-shaped cells as shown in the pictures of Figure 19 taken from microscopic examination. These cells can be observed in any biological tissue. These latter were originally Ca II cells that underwent modifications, by extending themselves. At this stage it is not difficult to find splitting cells.

a b
c d
e f
Figure 19

The club-shaped cells, typical of the step Ca III, are universally present in all patients and in all solid tumors. They cannot be confused with the other cells of the organism because they do not belong to any human tissue. They are the precursors of the tumoral mass. In case the tumoral mass is not present yet at the moment of the test it will appear in the following months.

While in Orgonomy, the developments and the steps towards an oncological pathology can be diagnosed many years in advance by detecting and monitoring the presence of the Ca I, Ca II, and Ca III cells, and of the thorny red blood cells, traditional oncology, in the same conditions, is not able to do a reliable cancer diagnosis in that does not see and recognize any cancer cell (and the presence of the tumoral mass).

The step 4 (Ca IV) is characterized by the presence of ameboid, mobile cells. Figure 20 shows an example of a Ca IV mobile cell taken from sputum of a terminally-ill male patient with a lung cancer.

Figure 20

These Ca IV cells are no longer belonging to those cells of the phase 1 that allow an earlier cancer diagnosis to be done. Indeed, when they appear, the cancer process is in the phase 2 and is already well advanced. At this point cancer can be also diagnosed by the standard methods of classical oncology.

The Ca V cells represent the last step of a terminally-ill patient. In a sample of a patient at this stage all the cells of the different steps above-described can be found. Being an ongoing process, we can find Ca I cells in vesicular disintegration, bionous re-organization into Ca II cells, club-shaped Ca III cells, T-bacilli, all available in a mush or mixture that does not give much hopes to the patient.

Figure 21 shows blood cells and tissues in Ca V taken from the blood of a female dog operated on for breast cancer.

Figure 21

According to the results above reported and obtained by examining at the microscope biological samples of healthy and cancer-ill patients, we were able to ascertain that blood and cells in a bioenergetically-weak organism are morphologically different and distinguishable from those of a bioenergetically-charged organism.

Red blood cells in the blood, too, show two different reactions according to the energy level of the organism.

Bioenergetically-charged red blood cells at the microscope examination exhibit more and more large and turgid bluish vesicles inside the stroma that appear like pearls nestled around a ring. While, bioenergetically-weak red blood cells are instead much smaller, more wrinkled and thorny. As far as cells are concerned, they undergo very clear morphological modifications that reflect the level of the proper bioenergetic charge.

The first signal is the vesicular reaction of the whole cell. In this first step the cytoplasm of the weaker cells fill up of small vesicles (bions) and sometimes even of T-bacilli. Such cells, belonging to the Ca I step, represent the first indication and warning of a scarce or low energetic charge. Over time, being as these vesicles are active, they aggregate and merge themselves thus forming new conglomerates and structures that are surrounded by a membrane. Cells found in this condition are belonging to the Ca II step.

These structures further develop forming oblong cells that Reich called club-shaped cells, belonging to the Ca III step of the full process. Ca III cells are alien to the organism, they do not belong to any human tissue and anticipate the formation of the tumoral mass.

Figure 22 shows pictures of possible cells that can be found when examining at the microscope biological samples of a cancer patient.

Figure 22

The following Figure 23 represents a comparison between pictures of cells that can be found in a healthy organism and in a cancer patient.

Figure 23

The results so far obtained in more than three decades of in-house microscope examinations on a significant number of patients or pets strikingly confirmed Reich’s findings and assumptions. All the modifications observed at the microscope related to the red blood cells and the cells in urinary sediments or sputum occurred exactly as Reich described and reported in his writings. Examinations confirmed that Ca I and Ca II cells appear much in advanced of the time the tumor is detectable.

These cells are the first sign of an ongoing cancer process and can be detected even decades earlier than the actual insurgence of the tumoral mass.

Ca III cells are the precursors of the tumoral mass that in some cases is already present at the time of the test while in other cases can be found some months later.

As above mentioned, the procedure that Reich developed for an early diagnosis of cancer is valid for all solid tumors.

This evidences the fact that all tumoral cells have the same origin and, as a consequence, all kinds of known cancers are indeed one and the same disease. It has also been determined that the process is universal and occurs similarly both in female and male human organisms, independent of age, and similarly, in animals.

The author wishes to thank Roberto Maglione and Leon Southgate for their suggestions in writing the paper.


  1. Reich W, The Cancer Biopathy. Volume II of the Discovery of the Orgone, Farrar Straus & Giroux, New York, 1973; see also Reich W, "Cancer Cells" in Experiment XX, Orgone Energy Bulletin, Vol 3, N° 1, January 1951, Orgone Institute Press, Rangeley, Usa
  2. Reich W, Bion Experiments on the Cancer Problem (dated July 1939), Orgonomic Functionalism, Usa, Volume 7, Spring 2019, page 38
  3. Reich W, Ibid, page 52


Armando Vecchietti

Vecchietti has been studying Reich’s theories since the early 1970’s, and has been doing research on cancer cell development and the Reich blood test for more than 40 years. He is performing routinely Reich blood test for the early diagnosis of cancer in his private practice. Vecchietti presented the results of his investigations in many Conferences both in Italy and abroad, and wrote many papers on the above subjects. He holds a degree in biology taken at the University of Camerino, Italy, in 1977.

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